Experimental Models for Renal Diseases: Pathogenesis and Diagnosis (Contributions to Nephrology, Vol. 169) - kelloggchurch.org

Experimental Models for Renal DiseasesPathogenesis and.

Jan 14, 2011 · Experimental Models for Renal Diseases: Pathogenesis and Diagnosis Contributions to Nephrology, Vol. 169: 9783805595377: Medicine & Health Science Books. Experimental Models for Renal Diseases Pathogenesis and Diagnosis ISBN: 978-3-8055-9537-7 e-ISBN: 978-3-8055-9538-4 DOI: 10.1159/isbn.978-3-8055-9538-4 X372 p., 76 fig., 27 in color, 13 tab., hard cover, 2011 Status: available Notes.

Diabetes and the Kidney: TOC: Vol. 169, 2011: Experimental Models for Renal Diseases: TOC: Vol. 168, 2011: Hemodiafiltration - A New Era. Pathophysiology of Renal Disease: TOC: Vol. 22, 1980: Vitamin D and Calcium Metabolism in Renal Diseases: TOC:. Contributions to Nephrology Abbreviation: Contrib Nephrol ISSN: 0302-5144 Print. Feb 19, 2018 · In this Review, the authors discuss animal models of various human immunoglobulin deposition diseases, and each model's drawbacks and contributions to our understanding of these diseases. Objective. A retrospective analysis of clinical characteristics and immunological manifestations of primary Sjogren’s syndrome pSS patients with or without renal involvement was conducted in order to elucidate the potential risk factors of renal damage in pSS and evaluate the condition. Methods. A total of 1002 patients, who fulfilled the 2002 classification criteria for pSS from the. In numerous experimental models of renal disease, progression is accelerated in male animals. This disparity in the course of renal disease is replicated by hormonal manipulation, suggesting that the actions of sex hormones, rather than structural differences between the sexes, are responsible for gender dimorphism in renal disease progression. The authors discuss experimental models that have helped elucidate systems involved in human disease, recent advances in this field as well as new therapies and future challenges.

Breakdown of chronic kidney disease CKD by stage 1–5 in selected countries with data available from the 2000s.9, 18, 19, 20, 21, 22, 23 Stage 3 corresponds to a. The nuanced and highly interdependent relationship between the kidney and the heart was described as early as 1836 by Robert Bright, who outlined the significant cardiac structural changes seen in patients with advanced kidney disease. 1 Since then, numerous advances have been made in summarizing the cardiorenal link in terms of hemodynamic phenotypes, pathophysiology, therapeutic options, and. Mar 01, 2011 · Pathophysiology of Acute Kidney Injury. and transition from pre-existing CKD to end-stage renal disease. Data indicate a close interaction between the many cell types involved in the. Apr 30, 2015 · 1. Introduction. Diabetes has long been a growing epidemic in the United States US and around the world. In 2011, there were 20.8 million people aged 18 years and older who carried a diagnosis of diabetes in the US alone [].The number of adults aged 18–79 in the US that were newly diagnosed with diabetes has more than tripled from 493,000 in 1980 to over 1.5 million in 2011 []. Jan 15, 2012 · High intraglomerular pressure is associated with renal hyperfiltration, leading to the initiation and progression of kidney disease in experimental models of diabetes mellitus DM. In humans, hyperfiltration is observed in patients with type 1 and type 2 DM, and is also seen in patients with pre-diabetic conditions, such as the metabolic syndrome.

Interstitial fibrosis is a common feature of chronic kidney disease, regardless of the etiology of the primary renal syndrome. Moreover, interstitial fibrosis is the strongest morphologic predictor of clinical outcome and is most tightly linked to progression of disease, even though the primary disease may be of glomerular origin. Detailed and sophisticated studies of experimental models of both spontaneous and induced disease of laboratory animals have repeatedly paved the way for a fuller appreciation of the potential immunopathogenetic mechanisms of glomerular and tubulointerstitial renal disease and have broadened understanding of the inflammatory process itself. Abnormalities in vitamin D metabolism play a major role in the pathogenesis of secondary hyperparathyroidism in chronic kidney disease. The gradual and progressive decline in 1,25-dihydroxyvitamin D in the course of chronic kidney disease is the result of several mechanisms that limit the ability of the failing kidney to maintain the levels of 1,25-dihydroxyvitamin D despite increasing.

High-Risk Indicators of Renal Involvement in Primary.

Jan 18, 2018 · Nature Reviews Nephrology, Vol. 15, Issue. 9, p. 559. its potential use as a biomarker as well as its role in the pathogenesis of renal and urinary diseases. a marker to predict ischemic brain damage in a rat stroke experimental model. Journal of. Oct 01, 2014 · C. Ronco Ed., Experimental Models for Renal Diseases: Pathogenesis and Diagnosis Herrera GA ed, Contributions to Nephrology Serieswith some changes, Vol. 169, Karger Pub, Basel, Switzerland 2011, pp. 232-244.

Glomerular basement membrane disorders in experimental models for renal diseases: Impact on understanding pathogenesis and improving diagnosis. / Kashtan, Clifford E.; Segal, Yoav. Experimental Models for Renal Diseases: Pathogenesis and Diagnosis. ed. / Guillermo Herrera. 2011. p. 175-182 Contributions to Nephrology; Vol. 169.

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