Genetic Factors in Atherosclerosis: Approaches and Model Systems (Monographs in Human Genetics, Vol. 12) -


Aug 30, 1989 · Genetic Factors in Atherosclerosis: Approaches and Model Systems Monographs in Human Genetics, Vol. 12 [A.J. Lusis, R.S. Sparkes, M. Schmid] on. FREE shipping on qualifying offers. Cardiovascular medicine. Atherosclerosis, the primary cause of coronary artery disease CAD and stroke, is a disorder with multiple genetic and environmental contributions. Genetic-epidemiologic studies have identified a surprisingly long list of genetic and nongenetic risk factors for CAD. However, such studies indicate that family history is the most significant independent risk factor ,. Many Mendelian. Genetic factors in atherosclerosis: approaches and model systems. [Robert S Sparkes; Aldons J Lusis;]. Monographs in human genetics, v. 12. Responsibility: volume editor, Aldons J. Lusis, Robert S. Sparkes. Reviews. User-contributed reviews Tags. Add tags for "Genetic factors in. the genetic investigations, the effects of environmental factors such as high cho-lesterol and fat or salt in the diet can be evaluated, so that the interactions be-tween environment and genotype can be assessed. Inwhatfollows, wedescribe our experiences while using genetargeting to study the genetics of atherosclerosis and hypertension.

Sep 14, 2000 · Atherosclerosis, a disease of the large arteries, is the primary cause of heart disease and stroke. In westernized societies, it is the underlying. Risk factors of atherosclerosis: A review of genetic epidemiology data from a Serbian population Article PDF Available in Experimental and clinical cardiology 112:78-82 · July 2006 with 65 Reads. New methodology using molecular and biochemical techniques together with novel genetic-statistical approaches are used. Genetic Factors in Atherosclerosis: Approaches and Model Systems. Monographs in Human Genetics, vol. 12. Karger, Basel. March 24, 2020: Vol. 141, Issue 12. March 17, 2020: Vol. 141, Issue 11. This approach has yielded several valuable models of atherosclerosis and hypertension. developments in the technology of molecular genetics have dramatically enhanced our capacity to study the influence of genetic factors on human vascular disease as well as to.

Volume 263, number 1 FEBS LETTERS April 1990 Booklist no. 94 Ammirato, P.V. et al. eds Handbook of plant cell culture, vol. 5. Genetic factors are currently recognized as major determinants of this pathology Galton and Ferns 1989, which is considered the most prevalent genetic disorder affecting humans Funke and Assman 1999. Susceptibility to atherosclerosis is believed to have a complex phenotype probably involving a number of linkages, and this complexity has. Atherosclerosis, the primary cause of coronary heart disease CHD, involves numerous cell types and organs and a number of disparate physiological processes see Figure 1. 1–3 It is not surprising, then, that the genetic basis of CHD is complex. This complexity is clearly illustrated by studies of rodent models; for example, experiments with transgenic and gene-targeted mice have revealed. Apr 14, 2013 · The analysis of the mechanisms linking environmental factors to the genes in the determination of human health is of importance in the field of life sciences and biomedical research. Several studies have demonstrated that family history, genetics, dietary, and cultural habits have a main role in the onset of gallstones [ 75 – 77 ].

Atherosclerosis Nature.

The pace of discovery in the application of human genetics to atherothrombotic disease and its risk factors has been accelerating. A large number of genetic loci have been identified in association with myocardial infarction MI, coronary artery disease CAD, thrombotic events, lipid traits, and other circulating biomarkers related to atherothrombotic disease. Oct 01, 2016 · 1. Introduction. Sepsis is the dysregulated host response to an infection which leads to life-threatening organ dysfunction according to the new Sepsis-3 definition Singer et al., 2016, Seymour et al., 2016.It can result in 28 day mortalities of up to 60% Engel et al., 2007, Angus and Wax, 2001.Consequently, there is an urgent need for new therapies but results from recent large scale.

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